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Subjective improvement if any would most likely involve reduced pain signals and decreased swelling rather than structural tissue repair

Others have identified astrocytes as a target for the neuroprotective effects of UA, demonstrating effects on astrocytic glutathione and Nrf2, and providing further support for the involvement of UA in antioxidative processes 52

Although there are so many drivers of the astrocyte hypertrophic profile in MS, the mechanisms activated by the majority of them seem to be triggered by neuroinflammation and neurodegeneration